Contagion processes on networks, including disease spreading, information diffusion, or social behaviors propagation, can be modeled as simple contagion, i.e., as a contagion process involving one connection at a time, or as complex contagion, in which multiple interactions are needed for a contagion event. Empirical data on spreading processes, however, even when available, do not easily allow us to uncover which of these underlying contagion mechanisms is at work. We propose a strategy to discriminate between these mechanisms upon the observation of a single instance of a spreading process. The strategy is based on the observation of the order in which network nodes are infected, and on its correlations with their local topology: these correlations differ between processes of simple contagion, processes involving threshold mechanisms, and processes driven by group interactions (i.e., by "higher-order"mechanisms). Our results improve our understanding of contagion processes and provide a method using only limited information to distinguish between several possible contagion mechanisms.
Distinguishing Simple and Complex Contagion Processes on Networks / Cencetti, Giulia; Contreras, Diego Andrés; Mancastroppa, Marco; Barrat, Alain. - In: PHYSICAL REVIEW LETTERS. - ISSN 0031-9007. - 130:24(2023). [10.1103/physrevlett.130.247401]
Distinguishing Simple and Complex Contagion Processes on Networks
Mancastroppa, Marco;
2023-01-01
Abstract
Contagion processes on networks, including disease spreading, information diffusion, or social behaviors propagation, can be modeled as simple contagion, i.e., as a contagion process involving one connection at a time, or as complex contagion, in which multiple interactions are needed for a contagion event. Empirical data on spreading processes, however, even when available, do not easily allow us to uncover which of these underlying contagion mechanisms is at work. We propose a strategy to discriminate between these mechanisms upon the observation of a single instance of a spreading process. The strategy is based on the observation of the order in which network nodes are infected, and on its correlations with their local topology: these correlations differ between processes of simple contagion, processes involving threshold mechanisms, and processes driven by group interactions (i.e., by "higher-order"mechanisms). Our results improve our understanding of contagion processes and provide a method using only limited information to distinguish between several possible contagion mechanisms.File | Dimensione | Formato | |
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